http://www.cnr.it/ontology/cnr/individuo/prodotto/ID55838
IL-18 in autoimmunity (Articolo in rivista)
- Type
- Label
- IL-18 in autoimmunity (Articolo in rivista) (literal)
- Anno
- 2006-01-01T00:00:00+01:00 (literal)
- Alternative label
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- Boraschi D, Dinarello CA (literal)
- Pagina inizio
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- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#numeroVolume
- Rivista
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- Note
- ISI Web of Science (WOS) (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
- Diana Boraschi: Laboratory of Cytokines, Unit of Immunobiology, Institute of Biomedical Technologies, National Research Council, CNR, Area della Ricerca di S. Cataldo, via G. Moruzzi 1, I-56124 Pisa, Italy
Charles A. Dinarello: Division of Infectious Diseases, University of Colorado Health Sciences Center, Denver, CO 80262, USA (literal)
- Titolo
- IL-18 in autoimmunity (literal)
- Abstract
- IL-18 is among the cytokines responsible for immune-mediated pathologies and is probably one of
the factors that contribute to the pathogenesis of autoimmune diseases. Identification of the causes of uncontrolled
IL-18 production and activity in autoimmunity would allow for novel therapeutic targets to effectively block
autoimmune activation and inhibit concomitant tissue damage. IL-18 is produced mainly by
monocytes/macrophages in response to stimuli of viral/bacterial origin, its production being therefore one of the
effects of innate immunity initiated by host-pathogen interaction. In this review, we summarise the evidence
supporting both the effector and the pathogenic role of IL-18 in autoimmunity, and propose that the disturbed
mechanism of innate immunity, resulting from macrophage activation through innate immunity receptors
(TLR/IL-1R family), may be the basis of pathologically high levels of IL-18 production and activation. Unravelling
the mechanisms of IL-18 production and activity in autoimmune diseases will allow the identification of targets
for more effective therapeutic intervention. (literal)
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