http://www.cnr.it/ontology/cnr/individuo/prodotto/ID4590
NF-kB and virus infection: who controls whom. (Articolo in rivista)
- Type
- Label
- NF-kB and virus infection: who controls whom. (Articolo in rivista) (literal)
- Anno
- 2003-01-01T00:00:00+01:00 (literal)
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- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- Santoro M.G.(1,2); Rossi A.(2); Amici C.(2) (literal)
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- L'articolo รจ stato pubblicato da una rivista scientifica con Impact factor di 13,117 (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#numeroVolume
- Rivista
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- ISI Web of Science (WOS) (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
- 1-Department of Biology University of Rome Tor Vergata, Via della Ricerca Scientifica, 00133 Rome, Italy
2-Institute of Neurobiology and Molecular Medicine, CNR, 00133 Rome, Italy (literal)
- Titolo
- NF-kB and virus infection: who controls whom. (literal)
- Abstract
- Among the different definitions of viruses, `pirates of
the cell' is one of the most picturesque, but also one of
the most appropriate. Viruses have been known for a
long time to utilize a variety of strategies to penetrate
cells and, once inside, to take over the host nucleic
acid and protein synthesis machinery to build up their
own components and produce large amounts of viral
progeny. As their genomes carry a minimal amount of
information, encoding only a few structural and regulatory
proteins, viruses are largely dependent on their
hosts for survival; however, despite their apparent
simplicity, viruses have evolved different replicative
strategies that are regulated in a sophisticated manner.
During the last years, the study of the elaborate
relationship between viruses and their hosts has led to
the understanding of how viral pathogens not only are
able to alter the host metabolism via their signaling
proteins, but are also able to hijack cellular signaling
pathways and transcription factors, and control them
to their own advantage. In particular, the nuclear
factor-kB (NF-kB) pathway appears to be an attractive
target for common human viral pathogens. This
review summarizes what is known about the control
of NF-kB by viruses, and discusses the possible outcome
of NF-kB activation during viral infection,
which may benefit either the host or the pathogen. (literal)
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