http://www.cnr.it/ontology/cnr/individuo/prodotto/ID38422
A Novel Loss-of-Function LGI1 Mutation Linked to Autosomal Dominant Lateral Temporal Epilepsy (Articolo in rivista)
- Type
- Label
- A Novel Loss-of-Function LGI1 Mutation Linked to Autosomal Dominant Lateral Temporal Epilepsy (Articolo in rivista) (literal)
- Anno
- 2008-01-01T00:00:00+01:00 (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#doi
- 10.1001/archneur.65.7.939 (literal)
- Alternative label
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- Striano P; de Falco A; Diani E; Bovo G; Furlan S; Vitello L; Pinardi F; Striano S; Michelucci R; de Falco FA; Nobile C (literal)
- Pagina inizio
- Pagina fine
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#numeroVolume
- Rivista
- Note
- ISI Web of Science (WOS) (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
- Muscular
and Neurodegenerative Diseases
Unit, Institute \"G. Gaslini,\"
University of Genoa, Genoa,
Italy (Dr P. Striano) /
Department of Neurological
Sciences, Epilepsy Center,
Federico II University
(Drs P. Striano and S. Striano)
and Division of Neurology,
Loreto Nuovo Hospital
(Drs A. de Falco and
F. A. de Falco), Napoli, Italy /
CNR Institute of Neurosciences,
Section of Padova (Mss Diani,
Bovo, and Furlan and
Dr Nobile) and Department of
Biology, University of Padova
(Dr Vitiello), Padova, Italy and
Department of Neurosciences,
Ospedale Bellaria, Bologna, Italy
(Drs Pinardi and Michelucci). (literal)
- Titolo
- A Novel Loss-of-Function LGI1 Mutation Linked to Autosomal Dominant Lateral Temporal Epilepsy (literal)
- Abstract
- Background: Mutations responsible for autosomal dominant
lateral temporal epilepsy have been found in the leucine-
rich, glioma-inactivated 1 (LGI1) gene.
Objectives: To describe the clinical and genetic findings
in a family with autosomal dominant lateral temporal
epilepsy and to determine the functional effects of
a novel LGI1 mutation in culture cells.
Design: Clinical, genetic, and functional investigations.
Setting: University hospital and laboratory.
Patients: An Italian family with autosomal dominant
lateral temporal epilepsy.
Main Outcome Measure: Mutation analysis.
Results: A novel LGI1 mutation, c.365T?A (Ile122Lys),
segregating with the disease was identified. The mutant
Lgi1 protein was not secreted by culture cells.
Conclusion: Our data provide further evidence that mutations
in LGI1 hamper secretion of the Lgi1 protein,
thereby precluding its normal function. (literal)
- Prodotto di
- Autore CNR
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