http://www.cnr.it/ontology/cnr/individuo/prodotto/ID37404
Dopamine D2 receptor signaling controls neuronal cell death induced by muscarinic and glutamatergic drugs (Articolo in rivista)
- Type
- Label
- Dopamine D2 receptor signaling controls neuronal cell death induced by muscarinic and glutamatergic drugs (Articolo in rivista) (literal)
- Anno
- 2002-01-01T00:00:00+01:00 (literal)
- Alternative label
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- Bozzi Y., Borrelli E. (literal)
- Pagina inizio
- Pagina fine
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#numeroVolume
- Rivista
- Note
- ISI Web of Science (WOS) (literal)
- Titolo
- Dopamine D2 receptor signaling controls neuronal cell death induced by muscarinic and glutamatergic drugs (literal)
- Abstract
- Dopamine (DA), through D1/D2 receptor-mediated signaling, plays a major
role in the control of epileptic seizures arising in the limbic system.
Excitotoxicity leading to neuronal cell death in the affected areas is a
major consequence of seizures at the cellular level. In this respect,
little is known about the role of DA receptors in the occurrence of
epilepsy-induced neuronal cell death. Here we analyze the occurrence of
seizures and neurotoxicity in D2R -/- mice treated with the cholinergic
agonist pilocarpine. We compared these results with those previously
obtained with kainic acid (KA), a potent glutamate agonist. Importantly,
D2R -/- mice develop seizures at doses of both drugs that are not
epileptogenic for WT littermates and show greater neurotoxicity. However,
pilocarpine-induced seizures result in a more widespread neuronal death in
both WT and D2R -/- brains in comparison to KA. Thus, the absence of D2R
lowers the threshold for seizures induced by both glutamate and
acetylcholine. Moreover, the dopaminergic control of epilepsy-induced
neurodegeneration seems to be mediated by distinct interactions of D2R
signaling with these two neurotransmitters.
(literal)
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