http://www.cnr.it/ontology/cnr/individuo/prodotto/ID33577
How is the reactivity of laccase affected by single-point mutations? Engineering laccase for improved activity towards sterically demanding substrates (Articolo in rivista)
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- How is the reactivity of laccase affected by single-point mutations? Engineering laccase for improved activity towards sterically demanding substrates (Articolo in rivista) (literal)
- Anno
- 2011-01-01T00:00:00+01:00 (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#doi
- 10.1007/s00253-011-3240-4 (literal)
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C. Galli, P. Gentili, C. Jolivalt, C. Madzak, R. Vadalà (2011)
How is the reactivity of laccase affected by single-point mutations? Engineering laccase for improved activity towards sterically demanding substrates
in Applied microbiology and biotechnology; Springer-Verlag, Dordrecht (Paesi Bassi)
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- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- C. Galli, P. Gentili, C. Jolivalt, C. Madzak, R. Vadalà (literal)
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- http://www.springer.com/life+sciences/microbiology/journal/253 (literal)
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- Dipartimento di Chimica, Università 'La Sapienza' and IMC-CNR Sezione Meccanismi di Reazione,
P.le A. Moro 5, 00185 Rome, Italy
Laboratoire Charles Friedel, UMR 7223 CNRS/ENSCP,
11 rue P. et M. Curie, 75231 Paris, France
INRA, UMR 1319 Micalis, AgroParisTech, CBAI,
78850 Thiverval-Grignon, France (literal)
- Titolo
- How is the reactivity of laccase affected by single-point mutations? Engineering laccase for improved activity towards sterically demanding substrates (literal)
- Abstract
- In spite of its broad specificity among phenols,
Trametes versicolor laccase hardly succeeds in oxidizing
hindered substrates. To improve the oxidation ability of this
laccase towards bulky phenolic substrates, we designed a
series of single-point mutants on the basis of the amino-acid
layout inside the reducing substrate active site known from
the crystal structure of the enzyme. Site-directed mutagenesis
has addressed four phenylalanine residues in key
positions 162, 265, 332, and 337 at the entrance of the
binding pocket, as these residues appeared instrumental for
docking of the substrate. These phenylalanines were
replaced by smaller-sized but still apolar alanines. A double
mutant F162A/F332A was also designed. Measurement of
the oxidation efficiency towards encumbered phenols has
shown that mutant F162A was more efficient than the wildtype
laccase. The double mutant F162A/F332A led to 98%
consumption of bisphenol A in only 5 h and was more
efficient than the single mutants in the aerobic oxidation of
this bulky substrate. In contrast, lack of appropriate
hydrophobic interactions with the substrate possibly
depresses the oxidation outcome with mutants F265A and
F332A. One explanation for the lack of reactivity of mutant
F337A, supported by literature reports, is that this residue is
part of the second coordination shell of T1 Cu. A mutation
at this position thus leads to a drastic coordination shell
destabilization. Thermal stability of the mutants and their
resistance in a mixed water-dioxane solvent have also been
investigated. (literal)
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