http://www.cnr.it/ontology/cnr/individuo/prodotto/ID238437
Bioreactor transient exposure activates specific neurotrophic pathways in cortical neurons. (Articolo in rivista)
- Type
- Label
- Bioreactor transient exposure activates specific neurotrophic pathways in cortical neurons. (Articolo in rivista) (literal)
- Anno
- 2010-01-01T00:00:00+01:00 (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#doi
- 10.1007/s12217-008-9102-8 (literal)
- Alternative label
V. Zimmitti, E. Benedetti, V. Caracciolo, P. Sebastiani, S. Di Loreto. (2010)
Bioreactor transient exposure activates specific neurotrophic pathways in cortical neurons.
in Microgravity Science and Technology; Springer, London (Regno Unito)
(literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- V. Zimmitti, E. Benedetti, V. Caracciolo, P. Sebastiani, S. Di Loreto. (literal)
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- IFT-CNR
Università degli studi L'Aquila,
Sbarro University Phyladelfia USA (literal)
- Titolo
- Bioreactor transient exposure activates specific neurotrophic pathways in cortical neurons. (literal)
- Abstract
- Altered gravity forces might influence neuroplasticity and can provoke changes in biochemical
mechanisms. In this contest, neurotrophins have a pivotal role, particularly nerve growth factor (NGF) and
brain derived neurotrophic factor (BDNF). A suspension of dissociated cortical cells from rat embryos was
exposed to 24 h of microgravity before plating in normal adherent culture system. Expression and transductional signalling pathways of NGF and BDNF were
assessed at the end of maturational process (8-10 days
in vitro). Rotating wall vessel bioreactor (RWV) preexposition did not induce changes in NGF expression
and its high affinity receptor TrkA. On the contrary
both BDNF expression and its high affinity receptor TrkB were strongly up-regulated, inducing Erk-
5, but not Erk-1/2 activation and, in turn, MEF2C
over-expression and activation. According to our previous and present results, we postulate that relatively
short microgravitational stimuli, applied to neural cells during the developmental stage, exert a long time
activation of specific neurotrophic pathways. (literal)
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