http://www.cnr.it/ontology/cnr/individuo/prodotto/ID21245
Transgenic mice overexpressing the wild-type form of the HMGA1 gene develop mixed growth hormone/prolactin cell pituitary adenomas and natural killer cell lymphomas. (Articolo in rivista)
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- Label
- Transgenic mice overexpressing the wild-type form of the HMGA1 gene develop mixed growth hormone/prolactin cell pituitary adenomas and natural killer cell lymphomas. (Articolo in rivista) (literal)
- Anno
- 2005-01-01T00:00:00+01:00 (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#doi
- 10.1038/sj.onc.1208501 (literal)
- Alternative label
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- Fedele M; Pentimalli F; Baldassarre G; Battista S; Klein-Szanto AJ; Kenyon L; Visone R; De Martino I; Ciarmiello A; Arra C; Viglietto G; Croce CM; Fusco A. (literal)
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- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#numeroVolume
- Rivista
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#pagineTotali
- Note
- ISI Web of Science (WOS) (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
- Istituto di Endocrinologia ed Oncologia Sperimentale del CNR c/o Dipartimento di Biologia e Patologia Cellulare e Molecolare,
Universita` di Napoli 'Federico II', 80131 Naples, Italy; Kimmel Cancer Center, Jefferson Medical College, Philadelphia, PA 19107,
USA; Experimental Histopathology, Fox-Chase Cancer Center, Philadelphia, PA 19111, USA; Department of Pathology, Thomas
Jefferson University Hospital, Philadelphia, PA, USA; Istituto dei Tumori di Napoli Fondazione 'G. Pascale', via Semmola, 80131
Naples, Italy; 6NOGEC (Naples Oncogenomic Center) - CEINGE, Biotecnologie Avanzate, Naples, Italy (literal)
- Titolo
- Transgenic mice overexpressing the wild-type form of the HMGA1 gene develop mixed growth hormone/prolactin cell pituitary adenomas and natural killer cell lymphomas. (literal)
- Abstract
- Overexpression of HMGA1 proteins is a constant feature
of human carcinomas.Moreover , rearrangements of this
gene have been detected in several human benign tumors of
mesenchymal origin.To define the role of these proteins in
cell transformation in vivo, we have generated transgenic
mice overexpressing ubiquitously the HMGA1 gene.
These mice developed mixed growth hormone/prolactin
cell pituitary adenomas and natural killer (NK)-T/NK cell
lymphomas.The HMGA1-induced expression of IL-2 and
IL-15 proteins and their receptors may account for the
onset of these lymphomas.At odds with mice overexpressing
a wild-type or a truncated HMGA2 protein,
adrenal medullar hyperplasia and pancreatic islet cell
hyperplasia frequently occurred and no increase in body
size and weight was observed in HMGA1 mice.Take n
together, these data indicate an oncogenic role of the
HMGA1 gene also in vivo. (literal)
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