http://www.cnr.it/ontology/cnr/individuo/prodotto/ID194243
Early Basal Insulin Therapy Decreases New-Onset Diabetes after Renal Transplantation (Articolo in rivista)
- Type
- Label
- Early Basal Insulin Therapy Decreases New-Onset Diabetes after Renal Transplantation (Articolo in rivista) (literal)
- Anno
- 2012-01-01T00:00:00+01:00 (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#doi
- 10.1681/ASN.2011080835 (literal)
- Alternative label
Hecking, M.;Haidinger, M.;Doller, D.;Werzowa, J.;Tura, A.;Zhang, J.;Tekoglu, H.;Pleiner, J.;Wrba, T.;Rasoul-Rockenschaub, S.;Mu?lbacher, F.;Schmaldienst, S.;Druml, W.;Horl, W.H.;Krebs, M.;Wolzt, M.;Pacini, G.;Port, F.K.;Saëmann, M.D. (2012)
Early Basal Insulin Therapy Decreases New-Onset Diabetes after Renal Transplantation
in Journal of the American Society of Nephrology
(literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- Hecking, M.;Haidinger, M.;Doller, D.;Werzowa, J.;Tura, A.;Zhang, J.;Tekoglu, H.;Pleiner, J.;Wrba, T.;Rasoul-Rockenschaub, S.;Mu?lbacher, F.;Schmaldienst, S.;Druml, W.;Horl, W.H.;Krebs, M.;Wolzt, M.;Pacini, G.;Port, F.K.;Saëmann, M.D. (literal)
- Pagina inizio
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- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#url
- http://www.ncbi.nlm.nih.gov/pubmed/22343119 (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#numeroVolume
- Rivista
- Note
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
- Hecking, Haidinger, Doller, Werzowa, Schmaldienst, Druml, Horl, Saëmann: Department of Nephrology, Medical University of Vienna, Vienna, Austria /
Tura, Pacini: Metabolic Unit, Institute of Biomedical Engineering, National Research Council, Padova, Italy /
Zhang, Port: Arbor Research Collaborative for Health, Ann Arbor, MI, United States /
Tekoglu, Wrba: Department of Informatics, Medical University of Vienna, Vienna, Austria /
Pleiner: Department of Coordinating Center for Clinical Studies, Medical University of Vienna, Vienna, Austria /
Rasoul-Rockenschaub, Mu?lbacher: Department of Surgery, Medical University of Vienna, Vienna, Austria /
Krebs: Department of Endocrinology, Medical University of Vienna, Vienna, Austria /
Wolzt : Department of Clinical Pharmacology, Medical University of Vienna, Vienna, Austria (literal)
- Titolo
- Early Basal Insulin Therapy Decreases New-Onset Diabetes after Renal Transplantation (literal)
- Abstract
- No effective interventions to reduce risk for new-onset diabetes after transplantation (NODAT), a condition
associated with postoperative hyperglycemia and reduced patient and graft survival, have been established.
In this 1-year, proof-of-concept clinical trial, we randomly assigned 50 renal transplant recipients to
immediate-postoperative isophane insulin for evening blood glucose 0 mg/dl (treatment group) or
short-acting insulin and/or oral antidiabetic agents for blood glucose 0-250 mg/dl (standard-of-care
control group).We included only patients without a history of diabetes who received tacrolimus. By the third
postoperative evening, all patients in the treatment group had blood glucose 0 mg/dl and were subsequently
treated with basal insulin; during the first 3 weeks after transplantation, themean6SD daily insulin
dosage was 17611 IU/d. Among controls, 23 (92%) of 25 had blood glucose 0 mg/dl and 18 (72%) of 25
received standard-of-care antihyperglycemic treatment. Asymptomatic hypoglycemia occurred five times in
the treatment group and once in the control group. Throughout follow-up, the treatment group had 73%
lower odds of NODAT (odds ratio, 0.27) than the control group, and hemoglobin A1c was on average 0.38%
lower in the treatment group than the control group. Twelve months after transplantation, all patients in the
treatment group were insulin-independent, whereas 7 (28%) of 25 controls required antidiabetic agents. The
groups did not differ for insulin sensitivity, but the treatment group showed better b-cell function throughout
the 1-year follow-up. In conclusion, this study suggests regimens that include basal insulin significantly reduce
the odds for NODAT after renal transplantation, presumably via insulin-mediated protection of b cells. (literal)
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