http://www.cnr.it/ontology/cnr/individuo/prodotto/ID190436
GWAS META-ANALYSIS OF SERUM URATE CONCENTRATIONS AND GOUT IN DIVERSE POPULATIONS (Abstract/Comunicazione in atti di convegno)
- Type
- Label
- GWAS META-ANALYSIS OF SERUM URATE CONCENTRATIONS AND GOUT IN DIVERSE POPULATIONS (Abstract/Comunicazione in atti di convegno) (literal)
- Anno
- 2012-01-01T00:00:00+01:00 (literal)
- Alternative label
Kottgen A., Albrecht E., Teumer A., Vitart V., Krumsiek J., O'Seaghdha C., Ciullo M., Fox C., Caulfield M.,
Bochud M., Gieger C. (2012)
GWAS META-ANALYSIS OF SERUM URATE CONCENTRATIONS AND GOUT IN DIVERSE POPULATIONS
in ACR/ARHP Annual Meeting, Edimburgh, 12/11/2012
(literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- Kottgen A., Albrecht E., Teumer A., Vitart V., Krumsiek J., O'Seaghdha C., Ciullo M., Fox C., Caulfield M.,
Bochud M., Gieger C. (literal)
- Note
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
- Freiburg University Hospital, Freiburg, Germany
Helmholz Zentrum, Munich, Germany
University of Greifswald, Greifswald, Germany
MRC HGU,IGMM,University of Edinburgh, Edinburgh, UK
Massachussets General Hospital, Boston, USA
CNR IGB, Napoli, Italy
Havard Medical School, Boston USA
Queen Mary University of London, London, UK
University of Lausane, Lausane, Switzerland
John Hopkins Bloomberg School of Public Health, Baltimore, USA (literal)
- Titolo
- GWAS META-ANALYSIS OF SERUM URATE CONCENTRATIONS AND GOUT IN DIVERSE POPULATIONS (literal)
- Abstract
- Uric acid is the, biochemically active, final breakdown product of purine oxidation in humans.
Hyperuricemia, elevated levels of serum urate, can cause gout, the most prevalent inflammatory arthritis
in developed countries. Furthermore, increased levels of serum urate are associated with components of
the metabolic syndrome, such as obesity, high blood pressure and insulin resistance, type 2 diabetes and
with cardiovascular disease. The heritability of serum urate concentrations is estimated at 50%-70%.
Eleven loci identified by previous genome-wide association studies (GWAS) explain 5-6% of serum urate
variance, suggesting that additional loci remain to be identified.
We performed a meta-analysis of GWAS on serum urate concentrations among 48 studies with more than
140,000 participants of European ancestry, and on gout among >70,000 individuals (2,125 cases) in the
Global Urate Genetics Consortium (GUGC). Secondary analyses included, among others, stratification
by sex, pathway analyses, and look-ups of the associated loci in individuals of other ancestries as well as
with urate-correlated traits. Replication was performed in 32,813 independent samples.
Altogether, we identified and replicated 28 genome-wide significant SNPs associated with serum urate
concentrations, including 18 new loci. Nominal association with gout and fractional excretion of urate
was found for 17 and 10 of the urate GWAS SNPs respectively, with consistent directions of effect. Effect
sizes on serum urate levels were similar among individuals of Indian ancestry, African Americans and
Japanese individuals. The genes implicated highlight new biological pathways offering novel avenues into
the treatment and prevention of gout. (literal)
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