http://www.cnr.it/ontology/cnr/individuo/prodotto/ID168636
Neuroprotective actions of a histidine analogue in models of ischemic stroke (Articolo in rivista)
- Type
- Label
- Neuroprotective actions of a histidine analogue in models of ischemic stroke (Articolo in rivista) (literal)
- Anno
- 2007-01-01T00:00:00+01:00 (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#doi
- 10.1111/j.1471-4159.2006.04412.x (literal)
- Alternative label
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- Sung-Chun Tang; Thiruma V. Arumugam; Roy G. Cutler; Dong-Gyu Jo; Tim Magnus; Sic L. Chan; Mohamed R. Mughal; Richard S. Telljohann; Matthew Nassar; Xin Ouyang; Andrea Calderan; Paolo Ruzza; Andrea Guiotto; Mark P. Mattson (literal)
- Pagina inizio
- Pagina fine
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- Rivista
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#numeroFascicolo
- Note
- ISI Web of Science (WOS) (literal)
- Scopu (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
- Laboratory of Neurosciences, National Institute on Aging Intramural Research Program, Baltimore, Maryland, USA; Department of Neurology, Stroke Center, National Taiwan University Hospital, Tapei, Taiwan; College of Pharmacy, Sungkyunkwan University, Suwan, South Korea; Istituto di Chimica Biomolecolare, Sezione di Padova, Padova, Italy; Department of Neuroscience, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA. (literal)
- Titolo
- Neuroprotective actions of a histidine analogue in models of ischemic stroke (literal)
- Abstract
- Histidine is a naturally occurring amino acid with antioxidant
properties, which is present in low amounts in tissues
throughout the body. We recently synthesized and characterized
histidine analogues related to the natural dipeptide
carnosine, which selectively scavenge the toxic lipid peroxidation
product 4-hydroxynonenal (HNE). We now report that
the histidine analogue histidyl hydrazide is effective in
reducing brain damage and improving functional outcome in
a mouse model of focal ischemic stroke when administered
intravenously at a dose of 20 mg/kg, either 30 min before or
60 min and 3 h after the onset of middle cerebral artery
occlusion. The histidine analogue also protected cultured rat
primary neurons against death induced by HNE, chemical
hypoxia, glucose deprivation, and combined oxygen and
glucose deprivation. The histidine analogue prevented neuronal
apoptosis as indicated by decreased production of
cleaved caspase-3 protein. These findings suggest a therapeutic
potential for HNE-scavenging histidine analogues in
the treatment of stroke and related neurodegenerative conditions. (literal)
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