http://www.cnr.it/ontology/cnr/individuo/prodotto/ID50024
A novel point mutation in PMP22 gene in an Italian family with hereditary neuropathy with liability to pressure palsies. (Articolo in rivista)
- Type
- Label
- A novel point mutation in PMP22 gene in an Italian family with hereditary neuropathy with liability to pressure palsies. (Articolo in rivista) (literal)
- Anno
- 2007-01-01T00:00:00+01:00 (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#doi
- 10.1016/j.jns.2007.05.034 (literal)
- Alternative label
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- Muglia M; Patitucci A; Rizzi R; Ungaro C; Conforti FL; Gabriele AL; Magariello A; Mazzei R; Motti L; Sabadini R; Sprovieri T; Marcello N; Quattrone A. (literal)
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- Rivista
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- Note
- ISI Web of Science (WOS) (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
- Institute of Neurological Sciences, National Research Council, Mangone, Cosenza, Italy;Neurology Unit, Arcispedale S. Maria Nuova, Reggio Emilia, Italy,Institute of Neurology, University Magna Graecia, Catanzaro, Italy (literal)
- Titolo
- A novel point mutation in PMP22 gene in an Italian family with hereditary neuropathy with liability to pressure palsies. (literal)
- Abstract
- Hereditary neuropathy with liability to pressure palsies (HNPP) is an autosomal dominant inherited disorder characterized by recurrent sensory or motor dysfunction. In 85% of HNPP cases the genetic defect is a 1.4 Mb deletion on chromosome 17p11.2, encompassing the PMP22 gene. Point mutations in the PMP22 gene responsible for HNPP phenotypes are rare. We investigated a 17-years-old girl who led to our detecting a novel mutation in PMP22 gene. The mutation was also detected in her father and corresponded to a deletion of one tymidine at position 11 in exon2 (c.11delT). This novel mutation creates a shift on the reading frame starting at codon 4 and leads to the introduction of a premature stop at codon 6. (literal)
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