http://www.cnr.it/ontology/cnr/individuo/prodotto/ID49975
Monoamine oxidase-a genetic variations influence brain activity associated with inhibitory control: new insight into the neural correlates of impulsivity (Articolo in rivista)
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- Monoamine oxidase-a genetic variations influence brain activity associated with inhibitory control: new insight into the neural correlates of impulsivity (Articolo in rivista) (literal)
- Anno
- 2006-01-01T00:00:00+01:00 (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#doi
- 10.1016/j.biopsych.2005.07.027 (literal)
- Alternative label
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- Passamonti L; Fera F; Magariello A; Cerasa A; Gioia MC; Muglia M; Nicoletti G; Gallo O; Provinciali L; Quattrone A (literal)
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- http://www.sciencedirect.com/science/article/pii/S0006322305009169 (literal)
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- ISI Web of Science (WOS) (literal)
- PubMe (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
- From the Neurology Unit, Department of Neurosciences, University \"Politecnica delle Marche,\" Ancona (Passamonti, Provinciali); Institute of Neurological Sciences, National Research Council, Piano Lago di Magone, Cosenza (Fera, Magariello, Cerasa, Gioia, Muglia, Nicoletti, Gallo, Quattrone); and Institute of Neurology, University \"Magna Graecia,\" Catanzaro, Italy (Quattrone). (literal)
- Titolo
- Monoamine oxidase-a genetic variations influence brain activity associated with inhibitory control: new insight into the neural correlates of impulsivity (literal)
- Abstract
- BACKGROUND: Previous evidence has shown that genetic variations in the serotonergic system contribute to individual differences in personality traits germane to impulse control. The monoamine oxidase-A (MAO-A) gene, coding for an enzyme primarily involved in serotonin and noradrenaline catabolism, presents a well-characterized functional polymorphism consisting of a variable number of tandem repeats in the promoter region, with high-activity and low-activity variants. High-activity allele carriers have higher enzyme expression, lower amine concentration, and present higher scores on behavioral measures of impulsivity than low-activity allele carriers.
METHODS: We studied the relationship of this polymorphism to brain activity elicited by a response inhibition task (Go/NoGo task), using blood oxygenation level-dependent (BOLD) functional magnetic resonance imaging in 24 healthy men.
RESULTS: Direct comparison between groups revealed a greater BOLD response in the right ventrolateral prefrontal cortex (Brodmann's area [BA] 45/47) in high-activity allele carriers, whereas a greater response in the right superior parietal cortex (BA 7) and bilateral extrastriate cortex (BA 18) was found in low-activity allele carriers.
CONCLUSIONS: These data suggest that a specific genetic variation involving serotonergic catabolism can modulate BOLD response associated with human impulsivity. (literal)
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