http://www.cnr.it/ontology/cnr/individuo/prodotto/ID42394
Endocrine modulation, inhibition of ovarian development and hepatic alterations in rainbow trout exposed to polluted river water (Articolo in rivista)
- Type
- Label
- Endocrine modulation, inhibition of ovarian development and hepatic alterations in rainbow trout exposed to polluted river water (Articolo in rivista) (literal)
- Anno
- 2010-01-01T00:00:00+01:00 (literal)
- Alternative label
VIGANO' L., BENFENATI E., BOTTERO S., CEVASCO A., MANDICH A., MONTEVERDE M. (2010)
Endocrine modulation, inhibition of ovarian development and hepatic alterations in rainbow trout exposed to polluted river water
in Environmental pollution (1987)
(literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- VIGANO' L., BENFENATI E., BOTTERO S., CEVASCO A., MANDICH A., MONTEVERDE M. (literal)
- Pagina inizio
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- Rivista
- Note
- ISI Web of Science (WOS) (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
- Water Research Institute, National Council of Research, Brugherio, Milan, Italy
Mario Negri Institute, Laboratory of Environmental Chemistry and Toxicology, Milan, Italy
Department of Environmental, Experimental and Applied Biology, University of Genoa, Genoa, Italy (literal)
- Titolo
- Endocrine modulation, inhibition of ovarian development and hepatic alterations in rainbow trout exposed to polluted river water (literal)
- Abstract
- Under laboratory conditions, female rainbow trout were exposed to graded concentrations of water from
the River Lambro, a polluted tributary of the River Po, and to the effluent of a large wastewater treatment
plant which flows into the River Lambro. In field exposures, trout were held in cages in the River Po
upstream and downstream from the confluence of the River Lambro. After 10-day (laboratory) and 30-day
(laboratory and field) exposures, trout were examined for several chemical, biochemical and histological
endpoints. The results indicated that exposure to complex mixtures of chemicals, including estrogen
receptor agonists, aryl-hydrocarbon receptor agonists, and probably antiandrogens, had occurred. Exposure
altered the plasma levels of 17b-estradiol and testosterone, and some treatments also enhanced
the activity of hepatic ethoxyresorufin O-deethylase. Gonadal histology showed varying levels of degenerative
processes characterised by oocyte atresia, haemorrhages, melano-macrophage centres (MMCs),
and oogonia proliferation. Liver histology showed less severe effects. (literal)
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