NO signalling in cytokinin-induced programmed cell death (Articolo in rivista)

Type
Label
  • NO signalling in cytokinin-induced programmed cell death (Articolo in rivista) (literal)
Anno
  • 2005-01-01T00:00:00+01:00 (literal)
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#doi
  • 10.1111/j.1365-3040.2005.01355.x (literal)
Alternative label
  • Carimi F, Zottini M, Costa A, Cattelan I, De Michele R, Terzi M, Lo Schiavo F (2005)
    NO signalling in cytokinin-induced programmed cell death
    in Plant, cell and environment (Print); Blackwell Publishing, Oxford (Regno Unito)
    (literal)
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
  • Carimi F, Zottini M, Costa A, Cattelan I, De Michele R, Terzi M, Lo Schiavo F (literal)
Pagina inizio
  • 1171 (literal)
Pagina fine
  • 1178 (literal)
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#url
  • http://onlinelibrary.wiley.com/doi/10.1111/j.1365-3040.2005.01355.x/pdf (literal)
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#numeroVolume
  • 28 (literal)
Rivista
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#pagineTotali
  • 8 (literal)
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#numeroFascicolo
  • 9 (literal)
Note
  • ISI Web of Science (WOS) (literal)
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
  • stituto di Genetica Vegetale, Consiglio Nazionale delle Ricerche, Corso Calatafimi 414, 90129 Palermo, Italy; Dipartimento di Biologia, Università Degli Studi di Padova, Via G. Colombo 3, Padua I-35121, Italy (literal)
Titolo
  • NO signalling in cytokinin-induced programmed cell death (literal)
Abstract
  • Cell death can be induced by cytokinin 6-benzylaminopurine (BA) at high dosage in suspension-cultured Arabidopsis cells. Herein, we provide evidence that BA induces nitric oxide (NO) synthesis in a dose-dependent manner. A reduction in cell death can be observed when the cytokinin is supplemented with the NO scavenger 2-(4-carboxyphenyl)-4,4,5,5-tetramethylimidazoline-1-oxyl-3-oxide (cPTIO) or the nitric oxide synthase (NOS) inhibitors: 2-aminoethylisothiourea (AET) and NG-monomethyl-L-arginine (L-NMMA), which suggests that NO is produced via a NOS and is a signalling component of this form of programmed cell death. In BA-treated cells, mitochondrial functionality is altered via inhibition of respiration. This inhibition can be prevented by addition of either cPTIO or AET implying that NO acts at the mitochondrial level. (literal)
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