http://www.cnr.it/ontology/cnr/individuo/prodotto/ID26485
Phenotypes of naturally defective recb allele in Neisseria meningitidis clinical isolates (Articolo in rivista)
- Type
- Label
- Phenotypes of naturally defective recb allele in Neisseria meningitidis clinical isolates (Articolo in rivista) (literal)
- Anno
- 2002-01-01T00:00:00+01:00 (literal)
- Alternative label
Salvatore P., Bucci C., Pagliarulo C., Tredici M., Colicchio R., Cantalupo G., Bardaro M., Del Giudice L., Massardo D.R., Lavitola A., Bruni C.B., Alifano P (2002)
Phenotypes of naturally defective recb allele in Neisseria meningitidis clinical isolates
in Infection and immunity (Print)
(literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
- Salvatore P., Bucci C., Pagliarulo C., Tredici M., Colicchio R., Cantalupo G., Bardaro M., Del Giudice L., Massardo D.R., Lavitola A., Bruni C.B., Alifano P (literal)
- Pagina inizio
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- Questo articolo mette in evidenza il fenotipo dell'allele recB difettivo in natura in isolati clinici di Neisseria meningitidis (literal)
- Note
- ISI Web of Science (WOS) (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
- Dipartimento di Biologia e Patologia Cellulare e Molecolare, Università di Napoli, Italy
Centro di Endocrinologia ed Oncologia Sperimentale, CNR, Napoli, Italy
Dipartimento di Scienze Ambientali, Seconda Università di Napoli, Caserta, Italy
Dipartimento di Scienze e Tecnologie Biologiche e Ambientali, Università di Lecce, Italy
Istituto di Genetica e Biofisica, CNR, Napoli, Italy (literal)
- Titolo
- Phenotypes of naturally defective recb allele in Neisseria meningitidis clinical isolates (literal)
- Abstract
- Neisseria meningitidis strains belonging to the hypervirulent lineage ET-
37 and several unrelated strains are extremely UV sensitive. The phenotype
is consequent to the presence of a nonfunctional recBET-37 allele carrying
multiple missense mutations. Phenotypic analysis has been performed with
congenic menigococcal strains harboring either the wild-type recB allele
or the recBET-37 allele. Congenic recBET-37 meningococci, in addition to
being sensitive to UV, were defective both in repair of DNA lesions
induced by UV treatment and partially, in recombination-mediated
transformation. Consistently, the wild-type, but not the recBET-37, allele
was able to complement the Escherichia coli recB21 mutation to UV
resistance and proficiency in recombination. recBET-37 meningococci did
not exhibit higher frequencies of spontaneous mutation to rifampin
resistance than recB-proficient strains. However, mutation rates were
enhanced following UV treatment, a phenomenon not observed in the recB-
proficient counterpart. Interestingly, the results of PCR-based assays
demonstrated that the presence of the recBET-37 allele considerably
increased the frequency of recombination at the pilin loci. The main
conclusion that can be drawn is that the presence of the defective recBET-
37 allele in N. meningitidis isolates causes an increase in genetic
diversity, due to an ineffective RecBCD-dependent DNA repair and
recombination pathway, and an increase in pilin antigenic variation. (literal)
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