Hepatic and Cardiac Steatosis Are They Coupled? (Articolo in rivista)

Type
Label
  • Hepatic and Cardiac Steatosis Are They Coupled? (Articolo in rivista) (literal)
Anno
  • 2012-01-01T00:00:00+01:00 (literal)
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#doi
  • 10.1016/j.hfc.2012.06.010 (literal)
Alternative label
  • Bugianesi, E ; Gastaldelli, A (2012)
    Hepatic and Cardiac Steatosis Are They Coupled?
    in Heart failure clinics; Elsevier Science Inc., New York (Stati Uniti d'America)
    (literal)
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori
  • Bugianesi, E ; Gastaldelli, A (literal)
Pagina inizio
  • 663 (literal)
Pagina fine
  • 670 (literal)
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#url
  • http://www.ncbi.nlm.nih.gov/pubmed/22999247 (literal)
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#numeroVolume
  • 8 (literal)
Rivista
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#pagineTotali
  • 8 (literal)
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#numeroFascicolo
  • 4 (literal)
Note
  • ISI Web of Science (WOS) (literal)
Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
  • [ 1 ] Univ Turin, San Giovanni Battista Hosp, Dept Internal Med, Div Gastro Hepatol, I-10126 Turin, Italy [ 2 ] CNR, Inst Clin Physiol, I-56124 Pisa, Italy (literal)
Titolo
  • Hepatic and Cardiac Steatosis Are They Coupled? (literal)
Abstract
  • Cardiac and hepatic fat are associated with insulin resistance and impaired suppression of lipolysis, ultimately leading to lipotoxicity. In the heart the lipotoxic effect translates into an impairment of energetic and mechanical efficiency, whereas in the liver a fibrogenic response is favored by the abundance of inflammatory cells. These features precede, and likely contribute to, left ventricular overload and cardiac hypertrophy through mechanisms similar to the ones observed in the progression of liver damage in nonalcoholic fatty liver disease (NAFLD). Collectively these findings suggest the presence of complex and intertwined interrelationships between NAFLD, myocardial steatosis, and coronary artery disease. (literal)
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