The mitochondrial permeability transition from in vitro artifact to disease target (Articolo in rivista)

Type

• Prodotto della ricerca (Classe)
• Articolo in rivista (Classe)

Label

• The mitochondrial permeability transition from in vitro artifact to disease target (Articolo in rivista) (literal)

Anno

• 2006-01-01T00:00:00+01:00 (literal)

Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#doi

• 10.1111/j.1742-4658.2006.05213.x (literal)

Alternative label

• Paolo Bernardi: Alexandra Krauskopf; Emy Basso; Valeria Petronilli; Elizabeth Blachly-Dyson; Fabio Di Lisa; Michael A. Forte (2006)
  The mitochondrial permeability transition from in vitro artifact to disease target
  in FEBS journal. S (Online)
  (literal)

Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#autori

• Paolo Bernardi: Alexandra Krauskopf; Emy Basso; Valeria Petronilli; Elizabeth Blachly-Dyson; Fabio Di Lisa; Michael A. Forte (literal)

Pagina inizio

• 2077 (literal)

Pagina fine

• 2099 (literal)

Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#url

• http://onlinelibrary.wiley.com/doi/10.1111/j.1742-4658.2006.05213.x/abstract (literal)

Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#numeroVolume

• 273 (literal)

Rivista

• FEBS journal. S (Rivista)

Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#pagineTotali

• 23 (literal)

Note

• ISI Web of Science (WOS) (literal)

Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni

• Paolo Bernardi: Alexandra Krauskopf; Emy Basso; Valeria Petronilli: Department of Biomedical Sciences and CNR Institute of Neurosciences, University of Padova, Italy Elizabeth Blachly-Dyson; Michael A. Forte: Vollum Institute, L474, Oregon Health and Sciences University, Portland, OR, USA Fabio Di Lisa: Department of Biological Chemistry and CNR Institute of Neurosciences, University of Padova, Italy (literal)

Titolo

• The mitochondrial permeability transition from in vitro artifact to disease target (literal)

Abstract

• The mitochondrial permeability transition pore is a high conductance channel whose opening leads to an increase of mitochondrial inner membrane permeability to solutes with molecular masses up to about 1500 Da. In this r0eview we trace the rise of the permeability transition pore from the status of in vitro artifact to that of effector mechanism of cell death. We then cover recent results based on genetic inactivation of putative permeability transition pore components, and discuss their meaning for our understanding of pore structure. Finally, we discuss evidence indicating that the permeability transition pore plays a role in pathophysiology, with specific emphasis on in vivo models of disease. (literal)

Prodotto di

• Istituto di neuroscienze (IN) (Istituto)

Autore CNR

• PAOLO BERNARDI (Unità di personale esterno)
• VALERIA PETRONILLI (Unità di personale interno)
• FABIO DI LISA (Persona)

Incoming links:

Autore CNR di

• VALERIA PETRONILLI (Unità di personale interno)
• PAOLO BERNARDI (Unità di personale esterno)
• FABIO DI LISA (Persona)

Prodotto

• Istituto di neuroscienze (IN) (Istituto)

Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#rivistaDi

• FEBS journal. S (Rivista)