http://www.cnr.it/ontology/cnr/individuo/prodotto/ID178805
Protective role of carnosine after INOS induction by LPS and INF-gamma treatment in primary rat astroglial cellcultures (Abstract/Comunicazione in rivista)
- Type
- Label
- Protective role of carnosine after INOS induction by LPS and INF-gamma treatment in primary rat astroglial cellcultures (Abstract/Comunicazione in rivista) (literal)
- Anno
- 2006-01-01T00:00:00+01:00 (literal)
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- Anna Maria Giuffrida Stella; Vittoria Spina Purrello; Salvatrice Costantino; Cristian Cuppari; Giulia Grasso; Rosaria Di Mare; Davide Licciardello; Vita Maria Marino ;Anna Maria Santoro; Vincenzo Giuseppe Nicoletti;Enrico Rizzarelli. (literal)
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- Abstract (literal)
- ISI Web of Science (WOS) (literal)
- Http://www.cnr.it/ontology/cnr/pubblicazioni.owl#affiliazioni
- Anna Maria Giuffrida Stella; Vittoria Spina Purrello; Salvatrice Costantino; Cristian Cuppari; Rosaria Di Mare; Davide Licciardello; Vita Maria Marino ;Vincenzo Giuseppe Nicoletti;Enrico Rizzarelli.University of Catania, Department of Chemical Sciences,Biochemistry and Molecular Biology Section, Catania, (literal)
- Titolo
- Protective role of carnosine after INOS induction by LPS and INF-gamma treatment in primary rat astroglial cellcultures (literal)
- Abstract
- Carnosine, a b-alanyl-histidine dipeptide, is present in quite high
concentration in brain, it seems to exert a protective effect against
cell damage caused by oxidants. In the present study we investigated
the effect of carnosine addition (20 mM) against NO-induced
oxidative stress in rat astroglial cell cultures. We measured cell
viability (MTT), nitrite production, LDH release into culture
medium and the effect of a specific inhibitor of iNOS, the
N-mono-metil-arginine (NMMA 1 mM). After LPS (4 lg/ml) and
INF-c (400U/ml) treatment, a decrease of cell viability and an
increase of LDH and nitrite release was observed. Interestingly all
these effects were prevented by carnosine addition. Moreover, as
determined by ESI Mass spectra, carnosine was able to trap NO
forming NO-carnosine adducts. Furthermore an increase of cell
survival and a decrease of LDH and nitrite release were also
observed after NMMA addition demonstrating that oxidative stress
in these conditions is due to iNOS induction. The effects of
carnosine treatment in all the experimental conditions tested,
demonstrate a marked protection of this compound against
NO-induced oxidative stress. The data obtained strongly suggest
an antioxidant action of carnosine, however further investigations
are necessary to better clarify its role for possible clinical
application. (literal)
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