http://www.cnr.it/ontology/cnr/individuo/prodotto/ID15908
Glucosamine and its N-acetyl-phenylalanine derivative prevent TNF-alpha-induced transcriptional activation in human chondrocytes (Articolo in rivista)
- Type
- Label
- Glucosamine and its N-acetyl-phenylalanine derivative prevent TNF-alpha-induced transcriptional activation in human chondrocytes (Articolo in rivista) (literal)
- Anno
- 2008-01-01T00:00:00+01:00 (literal)
- Alternative label
D'Abusco A.S., Cicione C., Calamia V., Negri R., Giordano C., Grigolo B., Politi L., Scandurra R. (2008)
Glucosamine and its N-acetyl-phenylalanine derivative prevent TNF-alpha-induced transcriptional activation in human chondrocytes
in Clinical and Experimental Rheumatology (Testo stamp.)
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- D'Abusco A.S., Cicione C., Calamia V., Negri R., Giordano C., Grigolo B., Politi L., Scandurra R. (literal)
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- 1. Univ Roma La Sapienza, Dept Biochem Sci, I-00185 Rome, Italy
2. Univ Roma La Sapienza, Lab Funct Genom & proteom Model Syst, Rome, Italy
3. Univ Roma La Sapienza, CNR, Inst Biomol Chem, Rome, Italy
4. Univ Bologna, Inst Codivilla Putti, Lab Immunol & Genet, I-40126 Bologna, Italy (literal)
- Titolo
- Glucosamine and its N-acetyl-phenylalanine derivative prevent TNF-alpha-induced transcriptional activation in human chondrocytes (literal)
- Abstract
- Glucosamine (GlcN) is used in the treatment of osteoarthritis as symptomatic slow-acting drug, but its mode of action is not completely known. We analyzed the influence of GlcN and its N-acetyl-phenylalanine derivative (NAPA) on mRNA transcription level of TNF-alpha-stimulated genes in cell culture.
(literal)
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